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Obesity in children is rising dramatically, and it comes with major – and sometimes lifelong – health consequences

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Obesity in children is rising dramatically, and it comes with major – and sometimes lifelong – health consequences

Knowing how to to kids about healthful eating is key.
MI PHAM/Unsplash

Christine Nguyen, University of Southern California

In the past two decades, children have become more obese and have developed obesity at a younger age. A 2020 found that 14.7 million children and adolescents in the U.S. with obesity.

Because obesity is a known risk factor for serious health problems, its rapid increase during the COVID-19 pandemic raised alarms.

Without intervention, many obese adolescents will remain obese as adults. Even before adulthood, some children will have serious problems beginning in their preteen years.

To address these issues, in early 2023, the American Academy of Pediatrics released its first new obesity management guidelines in 15 years.

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I am a pediatric gastroenterologist who sees children in the largest public hospital in California, and I have witnessed a clear trend over the last two decades. Early in my practice, I only occasionally saw a child with a complication of obesity; now I see multiple referrals each month. Some of these children have severe obesity and several health complications that require multiple specialists.

These observations prompted my for the California Health Equity Fellowship at the University of Southern California.

It's important to note that not all children who carry extra weight are unhealthy. But evidence supports that obesity, especially severe obesity, requires further assessment.

How obesity is measured

The World Health Organization defines obesity as “abnormal or excessive fat accumulation that a risk to health.”

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Measuring fat composition requires specialized equipment that is not available in a regular doctor's office. Therefore most clinicians use body measurements to screen for obesity.

One method is body mass index, or BMI, a calculation based on a child's height and weight compared to age- and sex-matched peers. BMI doesn't measure body fat, but when BMI is high, it correlates with total body fat.

According to the American Academy of Pediatrics, a child qualifies as overweight at a BMI between the 85th and 95th percentile. Obese is defined as a BMI above the 95th percentile. Other screens for obesity include waist circumference and skin-fold thickness, but these methods are less common.

Because many children exceeded the limits of existing growth charts, in 2022 the Centers for Disease Control and Prevention introduced extended growth charts for severe obesity. Severe obesity occurs when a child reaches the 120th percentile or has a BMI over 35. For instance, a 6-year-old boy who is 48 inches tall and is 110 pounds would meet criteria for severe obesity because his BMI is 139th percentile.

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Severe obesity carries a heightened risk of liver disease, cardiovascular disease and metabolic problems such as diabetes. As of 2016, almost 8% of children ages 2 to 19 had severe obesity.

Other health problems associated with severe obesity include obstructive sleep apnea, bone and joint problems that can cause early arthritis, high blood pressure and kidney disease. Many of these problems occur together.

More and more children are developing diseases that have traditionally only been seen in adults.

How obesity affects the liver

The liver disease associated with obesity is called nonalcoholic fatty liver disease. To store excess dietary fat and sugar, the liver's cells fill with fat. Excess carbohydrates in particular get processed into substances similar to the breakdown products of alcohols. Under the microscope, a pediatric fatty liver looks similar to a liver with alcohol .

Occasionally children with fatty liver are not obese; however, the greatest risk factor for fatty liver is obesity. At the same BMI, Hispanic and Asian children are more susceptible to fatty liver disease than Black and white children. Weight reduction or reducing the consumption of fructose, a naturally occurring sugar and common food additive – even without significant weight loss – improves fatty liver.

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Fatty liver is the most common chronic liver disease in children and adults. In Southern California, pediatric fatty liver doubled from 2009 to 2018. The disease can progress rapidly in children, and some will have liver scarring after only a few years.

Although few children currently require liver transplants for fatty liver, it is the most rapidly increasing reason for transplantation in young adults. Fatty liver is the second-most common reason for liver transplantation in the U.S., and it will be the leading cause in the future.

A light micrograph image of fatty liver, with large vacuoles of triglyceride fat accumulated inside liver cells.
A light micrograph image of fatty liver, with large vacuoles of triglyceride fat inside liver cells.
Dr_Microbe/iStock via Getty Images Plus

Links between obesity and diabetes

Fatty liver is implicated in metabolic syndrome, a group of conditions that cluster together and increase the risk of cardiovascular disease and diabetes.

In a telephone interview, Dr. Barry Reiner, a pediatric endocrinologist, voiced his concerns to me about obesity and diabetes.

“When I started my practice, I had never heard of type 2 diabetes in children,” says Reiner. “Now, depending on which part of the U.S., between a quarter and a third of new cases of diabetes are type 2.”

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Type 1 diabetes is an autoimmune disease previously called juvenile-onset diabetes. Conversely, type 2 diabetes was historically considered an adult disease.

However, type 2 diabetes is increasing in children, and obesity is the major risk factor. While both types of diabetes have genetic and lifestyle influences, type 2 is more modifiable through diet and exercise.

By 2060, the number of people under 20 with type 2 diabetes will increase by 700%. Black, Latino, Asian, Pacific Islander and Native American/Alaska Native children will have more type 2 diabetes diagnoses than white children.

“The seriousness of type 2 diabetes in children is underestimated,” says Reiner. He added that many people express a misconception that type 2 diabetes is a mild, slow-moving disease.

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Reiner pointed to an important study showing that type 2 diabetes acquired in childhood can rapidly progress. As early as 10 to 12 years after their childhood diagnosis, patients developed nerve damage, kidney problems and vision damage. By 15 years after diagnosis, at an average age of 27, almost 70% of the patients had high blood pressure.

Most patients had more than one complication. Although rare, a few patients experienced heart attacks and strokes. When people with childhood onset diabetes became pregnant, 24% delivered premature infants, over double the rate in the general population.

Heart health

Cardiovascular changes associated with obesity and severe obesity can also increase a child's lifetime chance of heart attacks and strokes. Carrying extra weight at 6 to 7 years old can result in higher blood pressure, cholesterol and artery stiffness by 11 to 12 years of age. Obesity changes the structure of the heart, making the muscle thicken and expand.

Although still uncommon, more people in their 20s, 30s and 40s are having strokes and heart attacks than a few decades ago. Although many factors may contribute to heart attack and stroke, obesity adds to that risk.

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Talk about being healthy, not focusing on weight

Venus Kalami, a registered dietitian, spoke with me about the environmental and societal influences on childhood obesity.

“Food, diet, lifestyle and weight are often a proxy for something greater going on in someone's ,” says Kalami.

Factors beyond a child's control, including depression, access to healthy food and walkable neighborhoods, contribute to obesity.

Parents may wonder how to children without introducing shame or blame. First, conversations about weight and food should be age appropriate.

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“A 6-year-old does not need to be thinking about their weight,” says Kalami. She adds that even preteens and teenagers should not be focusing on their weight, though they likely already are.

Even “good-natured” teasing is harmful. Avoid diet talk, and instead discuss health. Kalami recommends that adults explain how healthy habits can improve mood, focus or kids' performance in a favorite activity.

“A 12-year-old isn't always going to know what is healthy,” Kalami said. “Help them pick what's available and make the best choice, which may not be the perfect choice.”

Any weight talk, either criticism or compliments for weight loss, may backfire, she adds. Praising a child for their weight loss can reinforce a negative cycle of disordered eating. Instead, cheer the child's better health and good choices.

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Dr. Muneeza Mirza, a pediatrician, recommends that parents model healthful behavior.

“Changes should be made for the whole ,” says Mirza. “It shouldn't be considered a punishment for that kid.”The Conversation

Christine Nguyen, 2023 California Health Equity Fellow, University of Southern California

This article is republished from The Conversation under a Creative Commons license. Read the original article.

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The Conversation

How to tell if a conspiracy theory is probably false

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theconversation.com – H. Colleen Sinclair, Associate Research Professor of Social Psychology, Louisiana – 2024-05-07 07:33:01

Conspiracy theories can muddle people's thinking.

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H. Colleen Sinclair, Louisiana State University

Conspiracy theories are everywhere, and they can involve just about anything.

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People believe false conspiracy theories for a wide range of reasons – including the fact that there are real conspiracies, like efforts by the Sackler to profit by concealing the addictiveness of oxycontin at the cost of countless American lives.

The extreme consequences of unfounded conspiratorial beliefs could be seen on the staircases of the U.S. Capitol on Jan. 6, 2021, and in the self-immolation of a protestor outside the courthouse holding the latest Trump trial.

But if hidden forces really are at work in the world, how is someone to know what's really going on?

That's where my research in; I'm a social psychologist who studies misleading narratives. Here are some ways to vet a claim you've seen or heard.

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An overview of a maze of passages between shrubs.

Sometimes there's nothing but the maze itself.

oversnap/E+ via Getty Images

Step 1: Seek out the evidence

Real conspiracies have been confirmed because there was evidence. For instance, in the allegations dating back to the 1990s that tobacco companies knew cigarettes were dangerous and kept that information secret to make money, scientific studies showed problematic links between tobacco and cancer. Court cases unearthed corporate documents with internal memos showing what executives knew and when. Investigative journalists revealed efforts to hide that information. explained the effects on their patients. Internal whistleblowers sounded the alarm.

But unfounded conspiracy theories reveal their lack of evidence and substitute instead several elements that should be red flags for skeptics:

  • Dismissing traditional sources of evidence, they are in on the plot.

  • Claiming that missing information is because someone is hiding it, even though it's common that not all facts are known completely for some time after an .

  • Attacking apparent inconsistencies as evidence of lies.

  • Overinterpreting ambiguity as evidence: A flying object may be unidentified – but that's different from identifying it as an alien spaceship.

  • Using anecdotes – especially vaguely attributed ones – in place of evidence, such as “people are saying” such-and-such or “my cousin's friend experienced” something.

  • Attributing knowledge to secret messages that only a select few can grasp – rather than evidence that's plain and clear to all.

Step 2: Test the allegation

Often, a conspiracy theorist presents only evidence that confirms their idea. Rarely do they put their idea to the tests of logic, reasoning and critical thinking.

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While they may say they do research, they typically do not apply the scientific method. Specifically, they don't actually try to prove themselves wrong.

So a skeptic can follow the method scientists use when they do research: Think about what evidence would contradict the explanation – and then go looking for that evidence.

Sometimes that effort will yield confirmation that the explanation is correct. And sometimes not. Like a scientist, ask yourself: What would it take for you to believe your perception was wrong?

A hand holds a magnifying glass over one silhouetted figure, which is connected in a diagram to other figures.

Look closely at allegations of massive conspiracies.

Boris Zhitkov/Moment via Getty Images

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Step 3: Watch out for tangled webs

When theories claim large groups of people are perpetrating wide-ranging activities over a long period of time, that's another red flag.

Confirmed conspiracies typically involve small, isolated groups, like the top echelon of a company or a single terrorist cell. Even the alliance among tobacco companies to hide their products' danger was confined to those at the top, who made decisions and enlisted paid scientists and ad agencies to spread their messages.

False conspiracies tend to implicate wide swaths of people, such as world leaders, mainstream outlets, the global scientific community, the Hollywood entertainment industry and interconnected government agencies.

The online manifesto of Max Azzarello – the man who self-immolated on the steps of a New York courthouse in April 2024– railed against a conspiracy allegedly including every president since Bill Clinton, sex offender Jeffrey Epstein, even the writers of “The Simpsons.”

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Remember that the more people who supposedly know a secret, the harder it is to keep.

Step 4: Look for a motive

Confirmed conspiracies tell stories about why a group of people acted as they did and what they hoped to gain. Dubious conspiracies involve a lot of accusations or just questions without examining what real benefit the conspiracy nets the conspirators, especially when factoring in the costs.

For instance, what purpose would NASA have to lie about the existence of Finland?

Be particularly suspicious when conspiracies allege an “agenda” being perpetrated by an entire sociodemographic, which is often a marginalized group, such as a “gay agenda” or “Muslim agenda.”

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Also look to see whether those spreading the conspiracy theories have something to gain. For example, scholarly research has identified the 12 people who are the primary sources of false claims about vaccinations. The researchers also found that those people profit from making those claims.

Step 5: Seek the source of the allegations

If you can't figure out who is at the root of a conspiracy allegation and thus how they came to know what they claim, that is another red flag. Some people say they have to remain anonymous because the conspiracists will take revenge for revealing information. But even so, a conspiracy can usually be tracked back to its source – maybe a social media account, even an anonymous one.

Over time, anonymous sources either forward or are revealed. For instance, years after the Watergate scandal took down Richard Nixon's presidency, a key inside source known as “Deep Throat” was revealed to be Mark Felt, who had been a high-level FBI official in the early 1970s.

Even the notorious “Q” at the heart of the QAnon conspiracy cult has been identified, and not by government investigators chasing leaks of national secrets. Surprise! Q is not the high-level official some people believed.

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Reliable sources are transparent.

A view of a person holding a flashlight standing in a dark field while a circular shape hovers overhead, beaming a light down.

This didn't happen.

David Wall/Moment via Getty Images

Step 6: Beware the supernatural

Some conspiracy theories – though none that have been proven – involve paranormal, alien, demonic or other supernatural forces. People alive in the 1980s and 1990s might remember the public fear that satanic cults were abusing and sacrificing children. That idea never disappeared entirely.

And around the same time, perhaps inspired by the TV “V,” some Americans began to believe in lizard people. It may seem harmless to keep hoping for evidence of Bigfoot, but the person who detonated a bomb in downtown Nashville on Dec. 25, 2020, apparently believed lizard people ran the Earth.

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The closer the conspiracy is to science fiction, the closer it is to just being fiction.

Step 7: Look for other warning signs

There are other red flags too, like the use of prejudicial tropes about the group allegedly behind the conspiracy, particularly antisemitic allegations.

But rather than doing the work to really examine their conspiratorial beliefs, believers often choose to write off the skeptics as fools or as also being in on it – whatever “it” may be.

Ultimately, that's part of the allure of conspiracy theories. It is easier to dismiss criticism than to admit you might be wrong.The Conversation

H. Colleen Sinclair, Associate Research Professor of Social Psychology, Louisiana State University

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This article is republished from The Conversation under a Creative Commons license. Read the original article.

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The Conversation

Future pandemics will have the same human causes as ancient outbreaks − lessons from anthropology can help prevent them

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theconversation.com – Ron Barrett, Associate Professor of Anthropology, Macalester College – 2024-05-07 07:33:36

The changes that came with the transition from foraging to farming paved the way for disease.

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Ron Barrett, Macalester College

The last pandemic was bad, but is only one of many infectious diseases that emerged since the turn of this century.

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Since 2000, the world has experienced 15 novel Ebola epidemics, the global spread of a 1918-like influenza strain and major outbreaks of three new and unusually deadly coronavirus infections: SARS, MERS and, of course, COVID-19. Every year, researchers discover two or three entirely new pathogens: the viruses, bacteria and microparasites that sicken and kill people.

While some of these discoveries reflect better detection methods, genetic studies confirm that most of these pathogens are indeed new to the human species. Even more troubling, these diseases are appearing at an increasing rate.

Despite the novelty of these particular infections, the primary factors that led to their emergence are quite ancient. Working in the field of anthropology, I have found that these are primarily human factors: the ways we feed ourselves, the ways we together, and the ways we treat one another. In a forthcoming book, “Emerging Infections: Three Epidemiological Transitions from Prehistory to the Present,” my colleagues and I examine how these same elements have influenced disease dynamics for thousands of years. Twenty-first century technologies have served only to magnify ancient challenges.

Neolithic infections

The first major wave of newly emerging infections occurred with the start of the Neolithic revolution about 12,000 years ago, when people began shifting from foraging to farming as their primary means of subsistence.

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Before then, human infections tended to be mild and chronic in nature, manageable burdens of long-term parasites that people carried around from place to place. But full-time agrarian living brought the kinds of acute and virulent infections that we are familiar with . This global shift was humanity's first epidemiological transition.

illustration of an Egyptian tomb engraving of farmers with domesticated animals

The first emerging infections followed the rise of intensive agriculture.

mikroman6/Moment via Getty Images

Farming itself was not the cause. Rather, it was the major lifestyle changes associated with this new enterprise. Agriculture supplied people with high-calorie grains, but often did so at the expense of dietary diversity, resulting in compromised immunity from nutritional deficiencies.

The human population increased dramatically, and so did the number of large and densely settled communities that could sustain the transmission of deadlier pathogens.

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Our ancient ancestors domesticated animals for food and labor, and their proximity to one another created opportunities for livestock diseases to evolve into human diseases.

Finally, the social hierarchies of newly agrarian societies led to disparities in the distribution of essential resources for healthy living.

These challenges of subsistence, settlement and social organization were the root causes of humanity's first major disease transition.

Declining infections

For a dozen millennia, these patterns spread across the world like a plague of plagues. They persisted until the 19th and 20th centuries, when expectancy rose with the precipitous decline of infectious diseases in high- and middle-income countries.

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Remarkably, the greatest proportion of this decline occurred before the discovery of effective antibiotics and most of the vaccines we use today. improvements were mainly due to nonmedicinal factors such as better farming and food distribution methods, major sanitation projects and housing reforms in poor urban areas.

Etching of unhygienic street conditions in 1800s New York City

Urban sanitation did more than new medicines to reduce infections in the 19th century.

Bettmann via Getty Images

These were significant reversals in the same ancient categories – subsistence, settlement and social organization – that led to the rise of infectious diseases in the first place. They resulted in humanity's second epidemiological transition, a significant but only partial reversal of the changes that first began in the Neolithic period.

This second pattern was not a panacea. Despite overall health improvements, chronic noninfectious conditions such as heart disease and cancer rose to become the primary causes of human mortality.

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Most low-income countries experienced a later version of this transition after World War II, but their health gains from declining infections were significantly less than those of their wealthier counterparts. At the same time, their losses to noninfectious diseases rose at comparable rates. These conflicting trends have led to a “worst-of-all-worlds” scenario with respect to the health of poor societies.

It is also worth noting that the declining infections in low-income societies have depended more on affordable antimicrobial drugs. Given the emergence of drug-resistant pathogens, these medicinal buffers are proving to be little more than short-term for the health consequences of poverty.

With the ability of pathogens to move freely across borders and boundaries, these consequences can quickly become everyone's problems.

part of Earth from space showing lines like flightpaths connecting cities

Every corner of the globe is connected by modern travel.

fotograzia/Moment via Getty Images

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Converging infections

In recent decades, humanity's interconnections have reached the point that nearly everyone now lives within a single global disease . Borders and boundaries no longer constrain the spread of distant outbreaks. The COVID-19 pandemic dramatically illustrated this new reality, when the SARS-CoV-2 virus spread around the world in only a few weeks.

The COVID-19 pandemic also highlighted the ways that infectious and noninfectious diseases can interact synergistically with one another to produce even worse outcomes than the simple sum of each disease. This is starkly illustrated by the majority of COVID-19 deaths, which occurred among people with chronic heart, lung and metabolic conditions that are common to a growing proportion of older people in populations both wealthy and poor.

When combined, these challenges have set the stage for the converging disease patterns visible today. This is the third epidemiological transition: the rise of new, virulent and drug-resistant infections occurring in a rapidly aging and highly interconnected world.

Unfortunately, the present pattern entails increasing outbreaks of new and deadly infections. The root causes of these outbreaks are in such as commercial agricultural practices, the urbanization of human populations and the challenges of poverty in the face of economic growth.

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Despite the magnitude of these determinants, they are essentially the same issues of subsistence, settlement and social organization from 12,000 years ago. Addressing these recurring issues will do more than prepare the world for future pandemics; it will to prevent them from in the first place.The Conversation

Ron Barrett, Associate Professor of Anthropology, Macalester College

This article is republished from The Conversation under a Creative Commons license. Read the original article.

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The Conversation

Venus is losing water faster than previously thought – here’s what that could mean for the early planet’s habitability

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theconversation.com – Eryn Cangi, Research Scientist in Astrophysical & Planetary Sciences, of Colorado Boulder – 2024-05-06 11:19:38
An artist's illustration of hydrogen disappearing from Venus.
Aurore Simonnet/ Laboratory for Atmospheric and Physics/ University of Colorado Boulder

Eryn Cangi, University of Colorado Boulder

Today, the atmosphere of our neighbor planet Venus is as hot as a pizza oven and drier than the driest desert on Earth – but it wasn't always that way.

Billions of years ago, Venus had as much water as Earth does today. If that was ever liquid, Venus may have once been habitable.

Over time, that water has nearly all been lost. Figuring out how, when and why Venus lost its water helps planetary scientists like me understand what makes a planet habitable — or what can make a habitable planet transform into an uninhabitable world.

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Venus, with clouds visible on its surface, photographed using UV light.
Venus, Earth's solar system neighbor.
JAXA/ISAS/DARTS/Kevin M. Gill, CC BY

Scientists have theories explaining why most of that water disappeared, but more water has disappeared than they predicted.

In a May 2024 study, my colleagues and I revealed a new water removal process that has gone unnoticed for decades, but could explain this water loss mystery.

Energy balance and early loss of water

The solar system has a habitable zone – a narrow ring around the Sun in which planets can have liquid water on their surface. Earth is in the middle, Mars is outside on the too-cold side, and Venus is outside on the too-hot side. Where a planet sits on this habitability spectrum depends on how much energy the planet gets from the Sun, as well as how much energy the planet radiates away.

The theory of how most of Venus' water loss occurred is tied to this energy balance. On early Venus, sunlight broke up water in its atmosphere into hydrogen and oxygen. Atmospheric hydrogen heats up a planet — like too many blankets on the bed in summer.

When the planet gets too hot, it throws off the blanket: the hydrogen escapes in a flow out to space, a process called hydrodynamic escape. This process one of the key ingredients for water from Venus. It's not known exactly when this process occurred, but it was likely within the first years or so.

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Hydrodynamic escape stopped after most hydrogen was removed, but a little bit of hydrogen was left behind. It's like dumping out a water bottle – there will still be a few drops left at the bottom. These leftover drops can't escape in the same way. There must be some other process still at work on Venus that continues to hydrogen.

Little reactions can make a big difference

Our new study reveals that an overlooked chemical reaction in Venus' atmosphere can produce enough escaping hydrogen to close the gap between the expected and observed water loss.

Here's how it works. In the atmosphere, gaseous HCO⁺ molecules, which are made up of one atom each of hydrogen, carbon and oxygen and have a positive charge, combine with negatively charged electrons, since opposites attract.

But when the HCO⁺ and the electrons react, the HCO⁺ breaks up into a neutral carbon monoxide molecule, CO, and a hydrogen atom, H. This process energizes the hydrogen atom, which can then exceed the planet's escape velocity and escape to space. The whole reaction is called HCO⁺ dissociative recombination, but we like to call it DR for short.

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Water is the original source of hydrogen on Venus, so DR effectively dries out the planet. DR has likely happened throughout the history of Venus, and our work shows it probably still continues into the present day. It doubles the amount of hydrogen escape previously calculated by planetary scientists, upending our understanding of present-day hydrogen escape on Venus.

Understanding Venus with data, models and Mars

To study DR on Venus we used both computer modeling and data analysis.

The modeling actually began as a Mars . My Ph.D. research involved exploring what sort of conditions made planets habitable for . Mars also used to have water, though less than Venus, and also lost most of it to space.

To understand martian hydrogen escape, I developed a computational model of the Mars atmosphere that simulates Mars' atmospheric chemistry. Despite being very different planets, Mars and Venus actually have similar upper atmospheres, so my colleagues and I were able to extend the model to Venus.

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We found that HCO⁺ dissociative recombination produces lots of escaping hydrogen in both planets' atmospheres, which agreed with measurements taken by the Mars Atmosphere and Volatile EvolutioN, or MAVEN, mission, a satellite orbiting Mars.

A spacecraft that looks like a metal box with two solar panels attached on either side and a small limb extending downward.
An illustration of the MAVEN mission orbiting Mars.
NASA's Goddard Space Flight Center

Having data collected in Venus' atmosphere to back up the model would be valuable, but previous missions to Venus haven't measured HCO⁺ – not because it's not there, but because they weren't designed to detect it. They did, however, measure the reactants that produce HCO⁺ in Venus' atmosphere.

By analyzing measurements made by Pioneer Venus, a combination orbiter and probe mission that studied Venus from 1978-1992, and using our knowledge of chemistry, we demonstrated that HCO⁺ should be present in the atmosphere in similar amounts to our model.

Follow the water

Our work has filled in a piece of the puzzle of how water is lost from planets, which affects how habitable a planet is for life. We've learned that water loss happens not just in one fell swoop, but over time through a combination of methods.

Faster hydrogen loss today via DR means that less time is required overall to remove the remaining water from Venus. This means that if oceans were ever present on early Venus, they could have been present for longer than scientists thought before water loss through hydrodynamic escape and DR started. This would more time for possible life to arise. Our results don't mean oceans or life were definitely present, though – answering that question will require lots more science over many years.

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There is also a need for new Venus missions and observations. Future Venus missions will provide some atmospheric measurements, but they won't focus on the upper atmosphere where most HCO⁺ dissociative recombination takes place. A future Venus upper atmosphere mission, similar to the MAVEN mission at Mars, could vastly expand everyone's knowledge of how terrestrial planets' atmospheres form and evolve over time.

With the technological advancements of recent decades and a flourishing new interest in Venus, now is an excellent time to turn our eyes toward Earth's sister planet.The Conversation

Eryn Cangi, Research Scientist in Astrophysical & Planetary Sciences, University of Colorado Boulder

This article is republished from The Conversation under a Creative Commons license. Read the original article.

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